Myocarditis in 15 year old – seeking suggestions
I feel sick to my stomach right now. My dear friend reluctantly had her 15 year old daughter vaxxed, under pressure from her ex, who is now in a relationship with a pro-vax primary physician. Turns out the dr didn’t end up vaxxing her own teen, but this one was already a done deal. Our teens go to a very small school together. Her daughter and another teen – both Vaxxed in August/September, both tested positive for Covid on Friday, and are symptomatic.
Yesterday and today, her daughter is complaining of a heavy pressure on her test. Her oxygen and other stats are good, but she is feeling this pressure that makes it hard to breathe. Friend just got a hold of her family doctor, who advised her that it is likely myocarditis. The doctor advised her to try ibuprofen to address the inflammation, and, if that does not help, to head to our local rural hospital for tests. My friend is choosing to head to the hospital now.
I have spent so much time reading about treatments for covid, and for vaccine injuries, but don’t recall reading any suggestions for myocarditis. I do recall the sobering facts and stats and percentages, so please spare me those, or other fear inducing info — but if anyone is aware of anything that might be able to address this proactively, or educate my friend on what steps she can possibly take, I would greatly appreciate that.
Unlike early treatment for Covid-19 itself, this is an issue that needs medical attention… glad to hear that your friend did not take a “wait-and-see” attitude. One small piece of advice would be to avoid, if at all possible, a Gadolinium injection as part of an imaging procedure. This can do further harm down the road… maybe one of the doctors here can chime in as to whether they think the benefits are worth the risks in the case of this procedure, in the context of a kid with prospective myocarditis.
I can only tell you that the Gadolinium imaging is not itself without risk;
Sorry to hear about the vaccinating. However, your friend has no jumped the gun and put the cart well before the horse. Nothing surprising that she is symptomatic and positive after getting vaxxed. But usually, most will get good protection for a couple months.
Here is where we have a problem. She is sick with covid and complaining about chest pain. Chest pain is a symptom of Covid. And that we are almost 6 weeks since vaccination, I do not believe it is pericarditis issue. This doctor to make this statement without any workup, unless they know vaccines are causing this, is just reckless. ( especially if they know the vaccines are causing this and not informing patients and still promoting the vax)
Then off to the hospital while with COVID. Its not like you can walk in and say hey , I got the vaccine, can you check and make sure I dont have pericarditis. They will laugh you right out of the hospital. But they wont, you have covid and even if you have heart stuff, its all COVID.. off to the COVID floor – limited visitation.. etc. RUNDEAthIS NEAR drug will be implemented. If you are lucky, they will take her vitals, and send her home to return if she gets worse…
My advice. Support her for COVID infection now. Forget anything about this heart stuff. She will likely take weeks just to rebound from COVID. If she is not suffering from irregular heart rhythms and not having pain in the chest ( when not sick with COVID) you do not need to see a cardiologist. if she has heart symptoms after recovering from COVID , and sees a cardiologist , they will not differentiate what may be from vaccine or from disease. You are not here yet to worry about this.. But based on the symptoms coming after infection, I would not be looking at the vaccine fault yet.
She is suffering from the inflammatory process that goes along with COVID-19 more than likely. If treated with a steroid and the FLCCC protocol, more than likely she can rebound rather quickly. FLCCC.net
Thank you all for your swift responses. You’ve helped me to stay out of panic. I have passed them on to the mom, who is appreciative. They held off on going to the hospital, per advice from the dad’s doctor girlfriend. I had already dropped off all the supplements on the preventative protocol. This time I suggested she look at the early treatment protocol and consider IVM which it turns out she has.
Having this doctor so close could be a blessing or not. I used to see her and found her too conventional for my preference – although generous with the time she spent with me. I guess it’s always nice to have a back channel to a doctor.
I would get her on the FLCCC.net recommended protocol for her situation asap.
Taurine is a powerful helper for the brain, heart, and kidneys. Benign supplement with no known toxic level. Crosses blood brain barrier. Can stop seizures. *Will drop blood sugar, so monitor. Good to take with food.
Physiological roles of taurine in heart and muscle
Stephen W Schaffer 1, Chian Ju Jong, K C Ramila, Junichi Azuma
Taurine (aminoethane sulfonic acid) is an ubiquitous compound, found in very high concentrations in heart and muscle. Although taurine is classified as an amino acid, it does not participate in peptide bond formation. Nonetheless, the amino group of taurine is involved in a number of important conjugation reactions as well as in the scavenging of hypochlorous acid. Because taurine is a fairly inert compound, it is an ideal modulator of basic processes, such as osmotic pressure, cation homeostasis, enzyme activity, receptor regulation, cell development and cell signalling. The present review discusses several physiological functions of taurine. First, the observation that taurine depletion leads to the development of a cardiomyopathy indicates a role for taurine in the maintenance of normal contractile function. Evidence is provided that this function of taurine is mediated by changes in the activity of key Ca2+ transporters and the modulation Ca2+ sensitivity of the myofibrils. Second, in some species, taurine is an established osmoregulator, however, in mammalian heart the osmoregulatory function of taurine has recently been questioned. Third, taurine functions as an indirect regulator of oxidative stress. Although this action of taurine has been widely discussed, its mechanism of action is unclear. A potential mechanism for the antioxidant activity of taurine is discussed. Fourth, taurine stabilizes membranes through direct interactions with phospholipids. However, its inhibition of the enzyme, phospholipid N-methyltransferase, alters the phosphatidylcholine and phosphatidylethanolamine content of membranes, which in turn affects the function of key proteins within the membrane. Finally, taurine serves as a modulator of protein kinases and phosphatases within the cardiomyocyte. The mechanism of this action has not been studied. Taurine is a chemically simple compound, but it has profound effects on cells. This has led to the suggestion that taurine is an essential or semi-essential nutrient for many mammals.
Query on PubMed. https://pubmed.ncbi.nlm.nih.gov/?term=taurine%20heart
Additionally CoQ10 has been used for heart failure. A study was done with heart failure patients that had been given 6 months to live. They gave 300mg/day and all, but one survived and improved. At the end, they stated they thought a better dose was 400mg/day
Coenzyme Q10 and Heart Failure
Heart failure (HF) with either preserved or reduced ejection fraction is associated with increased morbidity and mortality. Evidence-based therapies are often limited by tolerability, hypotension, electrolyte disturbances, and renal dysfunction. Coenzyme Q10 (CoQ10) may represent a safe therapeutic option for patients with HF. CoQ10 is a highly lipophilic molecule with a chemical structure similar to vitamin K. Although being a common component of cellular membranes, CoQ10’s most prominent role is to facilitate the production of adenosine triphosphate in the mitochondria by participating in redox reactions within the electron transport chain. Numerous trials during the past 30 years examining CoQ10 in patients with HF have been limited by small numbers and lack of contemporary HF therapies. The recent publication of the Q-SYMBIO randomized controlled trial demonstrated a reduction in major adverse cardiovascular events with CoQ10 supplementation in a contemporary HF population. Although having limitations, this study has renewed interest in evaluating CoQ10 supplementation in patients with HF. Current literature suggests that CoQ10 is relatively safe with few drug interactions and side effects. Furthermore, it is already widely available as an over-the-counter supplement. These findings warrant future adequately powered randomized controlled trials of CoQ10 supplementation in patients with HF. This state-of-the-art review summarizes the literature about the mechanisms, clinical data, and safety profile of CoQ10 supplementation in patients with HF.
Q10 and taurine great for the heart. But I want to add how essential magnesium is in that equation. And specifically magnesium taurate has been used successfully for arrhythmia. IV magnesium was found to be better than pharmaceuticals for controlling Afib in a canadian study.
In Q2, Pfizer’s revenues were up 86% operationally to $19bn – excluding sales of the BNT162b2 vaccine, revenues were still up 10% to $11bn.
Other notable gains in the quarter included rare disease medication Vyndaqel/Vyndamax, which was up by 77%.
Guess what Vyndamax is used to treat. You got it. Myocarditis.
You can’t make this shit up. And here’s the kicker.
Several heart failure physicians, specifically those who treat patients with cardiomyopathy caused by transthyretin amyloidosis (ATTR-CM), are calling attention to the extraordinarily high price of tafamidis and tafamidis meglumine (Vyndamax and Vyndaqel; Pfizer), both of which received orphan drug designation and were approved by the US Food and Drug Administration in 2019.
With a list price of $225,000 per year, tafamidis is the most expensive cardiovascular drug on the market.