mRNA and heart attack risk increase, an abstract

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  • Thu, Nov 25, 2021 - 02:01pm

    #1
    jjhw

    jjhw

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    mRNA and heart attack risk increase, an abstract

Abstract 10712: Mrna COVID Vaccines Dramatically Increase Endothelial Inflammatory Markers and ACS Risk as Measured by the PULS Cardiac Test: a Warning

https://www.ahajournals.org/doi/10.1161/circ.144.suppl_1.10712

This was actually covered by a MSM TV station here in the UK called GBNews-





  • Thu, Nov 25, 2021 - 02:29pm

    #2

    Jim H

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    This is a very important data set – it’s hard to overstate how important it is

I have written elsewhere today in the thread about IVM use in Japan of the importance of appreciating the importance of evidence stacks.  While it was clear to many of us that there was indeed a cause vs. effect relationship between the mRNA shots and cases of myocarditis, especially based on the temporal relationship that came out of deep analysis of relevant VAERS reports by biostatistician Dr. Jessica Rose.

Still, having a completely separate layer of evidence based on quantitative blood work from a large patient population against which a baseline exists is HUGE.  This is biochemical evidence of a different sort and it strengthens the case immensely.  It’s a suite of blood tests that are ALL elevating post double jab, each telling part of the same story.

It’s a story of pharma-hubris, of playing God with our immune systems without any appreciation for the downstream effects.  I actually handed a print out of the abstract to my doctor yesterday and told her once again (as if she didn’t know) that none of us will be getting the shots.

  • Thu, Nov 25, 2021 - 02:32pm

    #3

    Jim H

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    BTW, here is the data plotted out…

The ever-awesome Dr. John B took the time to plot the data from the abstract so that us visual learners could more readily grasp the magnitude of these effects;

  • Thu, Nov 25, 2021 - 02:59pm   (Reply to #3)

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    Oliveoilguy

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    mRNA and heart attack risk increase, an abstract

Jim H. Some questions:

Is the PULS test readily accessible by family physicians?

It seems to be a blood test. Does a person need a baseline from a previous PULS test in order for it to be instructive? Or is there some level that is valid for all people?

Has it been used in any studies by the vaccine manufacturers?

How reliable is this data from 566 subjects? Could it be a one off? Are there anymore statistics available?

  • Thu, Nov 25, 2021 - 03:46pm   (Reply to #2)

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    jjhw

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    mRNA and heart attack risk increase, an abstract

Jim H, I’m glad I could help, I have also sent the link to the abstract to Dr. John Campbell who will be covering it on his YT channel.

  • Thu, Nov 25, 2021 - 04:09pm   (Reply to #3)

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    Jim H

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    Attempting to answer Oliveoil…

Oh man.. lots of questions;

Is the PULS test readily accessible by family physicians?

The group that wrote the abstract are cardiologists…  but the tests and interpretation appear available to all.  You can read about it here;  https://pulstest.com/

Here’s an explanation of the intention of the test;

“The PULS (Protein Unstable Lesion Signature) Test measures the most clinically-significant protein biomarkers that measure the body’s immune system response to arterial injury.3

These injuries lead to the formation and progression of cardiac lesions which may become unstable and rupture, leading to cardiac event.”

It seems to be a blood test. Does a person need a baseline from a previous PULS test in order for it to be instructive? Or is there some level that is valid for all people?

Based on my reading of the abstract, there is a fixed interpretation of what the levels mean as correlating to overall risk, hence the pool of 566 patients had an average 11% 5-year risk of a cardiac event, which elevated to 25% post double jab.  The logical conclusion would be no baseline needed.  For the purposes of identifying a possible jab effect, the existence of a well sorted baseline made all the difference.

Has it been used in any studies by the vaccine manufacturers?

I would be shocked if they did.

How reliable is this data from 566 subjects? Could it be a one off? Are there anymore statistics available?

It would certainly be nice to have the raw data and put it in the hands of a capable biostatistician, but for now we have just a conference paper abstract.  566 is a BIG number of patients .. this is not at all trivial.  As I have stated in other threads discussing this abstract, I interpret the +/- values given as one standard deviation values… hence for instance in the case of the Hepatocyte Growth Factor (HGF) values, the one sigma ranges against the two means don’t even cross over.. so the data sets before and after seem very different.  Again it would be nice to have the raw data to calculate standard tests for differentiating data populations, but we don’t have that.

I did my own deep dive on HGF and what it means.  This paper says it very clearly;

https://www.cell.com/immunity/fulltext/S1074-7613(15)00223-X

Title:  HGF Guides T Cells into the Heart

T cells are the main determinants of adaptive immunity. Their presence in target tissue is regulated by the expression of distinct sets of adhesion and chemokine receptors and required to fulfill their immunological function. Naive and effector/memory T cells are distinguishable by their adhesion factor signatures: naive T cells can home only to lymph nodes (via CCR7, L-selectin, and the integrin LFA-1), whereas antigen-experienced T cells can travel to almost any tissue via many chemokine receptors and adhesion molecules (Ley, 2014, Mora and von Andrian, 2006). In order to return to the tissue where the antigen is present, there must be tissue tropism to allow a site-directed action of adaptive immunity……

…..Taken together, the results presented by Komarowska et al., 2015 introduce a new paradigm for cardiac T cell tropism. In addition, the data are exciting because they raise the potential to alter tissue-tropism of T cells that might modulate T cell-mediated pathology in the heart—and relevant therapeutic compounds are already available.

This elevation of just the HGF alone is very concerning.  It tells us that the spike protein, i.e. the “antigen” is present in the heart tissue of this patient population and that it is eliciting this HGF response, representative of a pathological condition in the heart.

We don’t even have to concern ourselves with the PULS overall score.. since that is interpolated.  HGF tells the story on it’s own.

  • Thu, Nov 25, 2021 - 04:38pm

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    sand_kitty

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    Reply To: mRNA and heart attack risk increase, an abstract

My understanding here is that 3 markers of vascular inflammation were found and correlated with risk of future heart attacks.  The main value of the PULS test is to inspire lifestyle changes.

Something similar was done with abdominal girth.  Those of us with big bellies are very likely insulin resistant.  So a simple tape measure around the waist lets you know if the patient in front of you is insulin resistant.  Not a perfect correlate, but pretty good.

My interpretation is that this study solidly documents vascular inflammation after vaccination.  This is big.  All the dying athletes now makes more sense at a mechanistic level.

But the test is not terribly useful except as a motivator for lifestyle change.

——

And I must take my hat off to our medical sleuths at PP.  I have learned tons of good stuff from Jim H and DaveF who are not officially in medicine.

  • Thu, Nov 25, 2021 - 06:35pm

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    jjhw

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    mRNA and heart attack risk increase, an abstract

Dr. John Campbells coverage-





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