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FROM NYC ICU: DOES COVID-19 REALLY CAUSE ARDS?

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  • Sun, Apr 05, 2020 - 05:58am

    #1
    shekinahl

    shekinahl

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    FROM NYC ICU: DOES COVID-19 REALLY CAUSE ARDS?

Is there any protocol (conventional as well as alternative) for rapid oxygenation of human cells?

  • Sun, Apr 05, 2020 - 01:26pm

    #2
    TurquoiseRose

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    Reply To: FROM NYC ICU: DOES COVID-19 REALLY CAUSE ARDS?

Acetazolamide, Nifedipine and Phosphodiesterase Inhibitors: Rationale for Their Utilization as Adjunctive Countermeasures in the Treatment of Coronavirus Disease 2019 (COVID-19) https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7096066/

Thought this was already posted- see this article as tables do not paste well.
Similar patterns of pulmonary disease between HAPE and COVID-19

HAPE: High altitude pulmonary edema; COVID-19: Coronavirus disease 2019;
In light of this, a countermeasure that has been shown to be effective in high altitude illness is Acetazolamide.

Sample medications and dosages utilized in high altitude illness and HAPE

HAPE: High altitude pulmonary edema
Acetazolamide  250 mg every 12 hours
Nifedipine 30 mg extended release every 12 hours
Sildenafil 20-50 mg every 8 hours
Tadalafil 10 mg every 12 hours

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Take away from video- using the wrong ventilator pressures will cause more harm than good with catastrophic iatrogenic lung injury.

This flawed Algorithm is widely circulated both nationally and internationally and is likely to be difficult to change.

He appears to be a maverick seeing harm and alerting us, as were the researchers publishing that Covid -19 was looking like HAPE and could perhaps be better served by trying meds that were already known to benefit HAPE (a failure of oxygenation).

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To tie this into Catherine Fitts -Plandemic, where she considers the possibility of  another variable as to why so many have flu symptoms.

A.   4-5G has been, by themselves are associated causing an increase in flu-like symptoms.

B.  5G has apparently also been associated with biological interference with oxygenation  https://www.takebackyourpower.net/critical-examination-of-the-coronavirus-5g-connection/

1) 60-GHz oxygen band: Precise experimental profiles and extended
absorption modeling in a wide temperature range [2011, Journal of Quantitative Spectroscopy & Radiative Transfer]
2) Evaluation of the Potential Biological Effects of the 60-GHz Millimeter Waves Upon Human Cells [2009, IEEE]
3) Fixed Wireless Communications at 60GHz Unique Oxygen Absorption Properties [2001, Shigeaki Hakusui, Harmonix Corporation]

  • Mon, Apr 06, 2020 - 09:55am

    #3
    TurquoiseRose

    TurquoiseRose

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    FROM NYC ICU: DOES COVID-19 REALLY CAUSE ARDS?

Covid-19 attacks the hemoglobin and may be mechanism causing poor oxygenation

±Covid- 19 Captures the Porphyrin to Inhibit Human Heme Metabolism
± ORF8 and surface glycoprotein could bind to the porphyrin

±orf1ab, ORF10, and ORF3a proteins could coordinate attack the heme on the 1-beta chain of hemoglobin to dissociate the iron to form the porphyrin
±attack will cause less +  less hemoglobin that can carry oxygen and carbon dioxide

±Extreme poisoning of lung  cells
±Inflammation due to the inability to exchange CO2 + O2 frequently resulting in ground-glass-like lung images
±Mechanism interferes with heme anabolic pathway = is expected to result in human disease

±Chloroquine could prevent orf1ab, ORF3a, and ORF10 from attacking heme to form porphyrin, + inhibit the binding of ORF8 and surface glycoproteins to porphyrins .
±This relieves symptoms of respiratory distress. Favipiravir could inhibit the envelope protein + ORF7a protein bind to porphyrin, prevent the virus from entering host cells, and catching free porphyrins.  See below Preprint:

https://chemrxiv.org/articles/COVID-19_Disease_ORF8_and_Surface_Glycoprotein_Inhibit_Heme_Metabolism_by_Binding_to_Porphyrin/11938173

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