AZ vax creator says covid-19 will wane “no place to go”
This article from today’s Times appears to contrast with Dr G vdB’s theory. Dr Gilbert is certainly qualified to speak on the topic. Let us hope she is correct.
Covid-19 will just end up causing a cold, says Oxford vaccine creator Sarah Gilbert
Chris Smyth, Whitehall Editor
Thursday September 23 2021, 12.01am BST, The Times
The new coronavirus is unlikely to mutate into a variant that can evade vaccines because there “aren’t very many places for the virus to go”, the creator of the Oxford jab has said.
Dame Sarah Gilbert, speaking yesterday in a Royal Society of Medicine webinar, played down fears of a more deadly new variant. “We normally see that viruses become less virulent as they circulate more easily and there is no reason to think we will have a more virulent version of Sars-CoV-2,” she said.She argued that the spike protein targeted by vaccines had only limited ability to mutate while still allowing the virus to get inside human cells. “If it changes its spike protein so much that it can’t interact with that receptor, then it’s not going to be able to get inside the cell. So there aren’t very many places for the virus to go to have something that will evade immunity but still be a really infectious virus,” she said.
It seems like the Delta variant has already proven that to be false.
I just got a text message from someone employed by a hospital on the west coast. Every medical provider in that hospital is vaccinated. But the disease is spreading rapidly through the nursing corps who have been sent home.
I also know that from the data coming out of Israel and Great Britain shows that there is little difference (percentage wise) in vaccinated/unvaccinated deaths. There is a difference in favor of the vaccinated, but it is not large.
I agree that the vaccines are currently failing due to the virus mutating. But that does not necessarily therefore mean that the virus can continue to mutate its spike indefinitely and still retain a functional spike that can still attach to the ACE2 receptors, which is what she is suggesting.
I do not know enough to know if this can be true but it is a point of view worth discussing from someone who should know what she is talking about.